Item type:Thesis, Open Access

Rolle des NOD-Signalings innerhalb des Makrophagen Lipidmetabolismus

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Philipps-Universität Marburg

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Abstract

NOD1 and NOD2 are a subset of Pattern Recognition Receptors (PRR), which can recognize pathogens based on their pathogen-associated molecular patterns (PAMPs) and thus regulate the innate immune response. Activation of NOD signaling leads to a complex inflammatory process, which is considered as the basis for the development of atherosclerosis and other diseases. Our group has already observed the influence of NOD1 and NOD2 on inflammation and atherogenesis. Based on these findings, this work investigated the expression of macrophages of wild-type mice after NOD stimulation with respect to cytokines, interleukins and other inflammatory mediators. Additionally, the influence of NOD stimulation on uptake/transport of cholesterol and foam cell formation were analyzed. This project shows that NOD stimulation of wild-type macrophages has a strong influence on the inflammatory process, as well as on the expression of scavenger receptors and efflux transporters. NOD stimulation led to an increased expression of the cytokines and interleukins. In addition, it was found that NOD stimulation caused an increased expression of the scavenger receptors such as SRA-1 and LOX-1. Also, a delayed induced ABCA-1 expression could be detected. In the second part of this work, two different methods were used to demonstrate that a systemic NOD1 and NOD2 deficiency leads to a reduction in foam cell formation. Furthermore, the influence of intravenous NOD activation on bone marrow cells, plasma and PBMCs was investigated. In summary, this research provides new insights into the role of NOD signaling within the macrophage with regard to atherogenesis and presents NOD receptors as essential mediators of inflammation. Further studies will be needed in the future to examine the role of NLRs in atherosclerosis in detail.

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Shahin, Kinan: Rolle des NOD-Signalings innerhalb des Makrophagen Lipidmetabolismus. : Philipps-Universität Marburg 2022-07-04. DOI: https://doi.org/10.17192/z2022.0211.