Item type:Thesis, Open Access

Einfluß der parenteralen Gabe von langkettigen Fettsäuren auf den Prostanoidstoffwechsel bei septischen Patienten

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Philipps-Universität Marburg

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Abstract

In this study the influence of Liposyn 20%, which is a fat emulsion containing polyunsaturated fatty acids, especially linoleic acid, on the eicosanoid metabolism of septic patients was examined, while given a total parenteral nutrition. On the septic intensive care unit of the general hospital Augsburg a short term and a long term period study were taken on septic patients. The short term period study took 4 days including 12 patients; the long term period study lasted 10 days including 6 patients. In each of these two stud-ies one half of the patients were fed exclusive a mixture of carbohydrates, while in the other group 50% of the non protein calories were provided by Liposyn 20%. Altogether the patients received each day 30 kcal/kg body weight as non protein calories.In addition in each group the patients were given 100g amino acids every day. To compare the patients regarding their severity of disease the APACHE III?Score was determined for each patient every day. In the eicosanoid laboratories of the childrens hospital of the university of Marburg these following index metabolites of the eicosanoids were measured using gas chromatography/mass spectrometry techniques: - 11-dehydro-TxB2 in plasma (measure for phasic TxA2-production), - TxB2 in 24h urin (measure for renal TxA2-production), - 2,3-dinor-TxB2 in 24h urin (measure for systemic TxA2-production), - 2,3-dinor-6-keto-PGF1a in 24h urin (measure for systemic PGI2- production), - PGE-M in 24h-urin (measure for systemic PGE2-production). In addition the contain of triglycerides and cholesterines in the plasma as well as the contain of linoleic acid in the cholesterinesters were determined. Regarding the APACHE?Score there was no difference in severity of disease between the examined patients. In the short term period study letality in the control group was little higher as in the group receiving fatty acids. In the long term period study there was no difference in letality between both groups. Triglycerides and cholesterines were normal, which shows a good elimination and metabolism of the given fat. All patients showed distinct increased levels of eicosanoids. Nor in the short term nor in the long term period study there was distinct difference in the eicosanoid production while given Liposyn 20%. The reason for this seems to be that during sepsis lipolysis is elevated which causes higher liberation of arachidonic acid out of the phospholipids of the cell membranes. This increase in liberated arachidonic acid resulted in an increased eicosanoid production in both groups. The additional supplementation of linoleic acid obviously didn?t led to a rise in arachidonic acid in the group given fatty acids, which can be explained by the rate limiting desaturases. Certainly if given over a longer period of time the supplementation of linoleic acid can conclude in a rise of arachidonic acid. It seems that the periods of time chosen in this study were obviously to short to result in this rise of arachidonic acid. The results of this study show that concerning the measured eicosanoids there are no reasons against the use of long chain polyunsaturated fatty acids as part of total parenteral nutrition for the duration of the examined period of time.In this study the influence of Liposyn 20%, which is a fat emulsion containing polyunsaturated fatty acids, especially linoleic acid, on the eicosanoid metabolism of septic patients was examined, while given a total parenteral nutrition. On the septic intensive care unit of the general hospital Augsburg a short term and a long term period study were taken on septic patients. The short term period study took 4 days including 12 patients; the long term period study lasted 10 days including 6 patients. In each of these two stud-ies one half of the patients were fed exclusive a mixture of carbohydrates, while in the other group 50% of the non protein calories were provided by Liposyn 20%. Altogether the patients received each day 30 kcal/kg body weight as non protein calories.In addition in each group the patients were given 100g amino acids every day. To compare the patients regarding their severity of disease the APACHE III?Score was determined for each patient every day. In the eicosanoid laboratories of the childrens hospital of the university of Marburg these following index metabolites of the eicosanoids were measured using gas chromatography/mass spectrometry techniques: - 11-dehydro-TxB2 in plasma (measure for phasic TxA2-production), - TxB2 in 24h urin (measure for renal TxA2-production), - 2,3-dinor-TxB2 in 24h urin (measure for systemic TxA2-production), - 2,3-dinor-6-keto-PGF1a in 24h urin (measure for systemic PGI2- production), - PGE-M in 24h-urin (measure for systemic PGE2-production). In addition the contain of triglycerides and cholesterines in the plasma as well as the contain of linoleic acid in the cholesterinesters were determined. Regarding the APACHE?Score there was no difference in severity of disease between the examined patients. In the short term period study letality in the control group was little higher as in the group receiving fatty acids. In the long term period study there was no difference in letality between both groups. Triglycerides and cholesterines were normal, which shows a good elimination and metabolism of the given fat. All patients showed distinct increased levels of eicosanoids. Nor in the short term nor in the long term period study there was distinct difference in the eicosanoid production while given Liposyn 20%. The reason for this seems to be that during sepsis lipolysis is elevated which causes higher liberation of arachidonic acid out of the phospholipids of the cell membranes. This increase in liberated arachidonic acid resulted in an increased eicosanoid production in both groups. The additional supplementation of linoleic acid obviously didn?t led to a rise in arachidonic acid in the group given fatty acids, which can be explained by the rate limiting desaturases. Certainly if given over a longer period of time the supplementation of linoleic acid can conclude in a rise of arachidonic acid. It seems that the periods of time chosen in this study were obviously to short to result in this rise of arachidonic acid. The results of this study show that concerning the measured eicosanoids there are no reasons against the use of long chain polyunsaturated fatty acids as part of total parenteral nutrition for the duration of the examined period of time.

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Zwingmann, Martin (12859733X): Einfluß der parenteralen Gabe von langkettigen Fettsäuren auf den Prostanoidstoffwechsel bei septischen Patienten. : Philipps-Universität Marburg 2003-12-22. DOI: https://doi.org/10.17192/z2003.0718.

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